Welcome to Part 2 – let's move beyond the acute setting to focus on the challenges of chronic low back pain, the impact of psychosocial factors (yellow flags), and how to recognise red flags that may signal serious underlying pathology.
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Welcome to Part 2 – let's move beyond the acute setting to focus on the challenges of chronic low back pain, the impact of psychosocial factors (yellow flags), and how to recognise red flags that may signal serious underlying pathology.
Not all pain is created equal – and understanding the neurophysiology behind acute and chronic pain is foundational for clinical practice.
Back pain is a common yet complex presentation in Australian primary care GP and ED. Understanding acute vs chronic pain, the underlying mechanisms, and evidence-based management strategies is key—especially when differentiating between specific and non-specific causes.
Here’s a clinical reasoning-based summary 👇
Back pain is common — but as a med student, your job isn’t just to recognise it. It’s to understand it. That means knowing the underlying pathophysiology, linking it to the history, and spotting red flags.
I've had a few student questions this week about exam technique so wanted to share my thoughts with all of you on how to best approach multiple choice / extended matching questions. Let me know what you think? 🤔🧠
Whether it’s a tickle in your throat or a full-blown coughing fit, this powerful reflex is all about protecting your lungs. But what's really going on behind the scenes?
In Part 1, we explored bronchodilators – which treat bronchoconstriction, one part of the asthma puzzle. But asthma is fundamentally a chronic inflammatory disease, and ongoing airway inflammation needs long-term control to prevent symptoms, reduce exacerbations, and preserve lung function.
I made this video 8 years or so ago about the role of aspirin sensitivity in asthma - the pharmacology is very cool- you might find it interesting? (it contains my favourite biochemical pathway ! )
Let’s look at the major bronchodilator classes used in asthma: short-acting β2 agonists (SABAs), long-acting β2 agonists (LABAs), and muscarinic antagonists (SAMAs and LAMAs) — and link them to the science behind the symptoms.